yellow phosphorus is a colorless wax-like crystals. Color in the light turn yellow, issued a garlic-like odor, atomic weight 30.97, vapor density 4.3kg/m3, melting point 44.1 ° C, boiling point 280 ° C, insoluble in water, soluble in organic solvents such as fats and carbon disulfide. Phosphorus can be spontaneous at room temperature, is easily oxidized into the oxide of phosphorus and phosphorus pentoxide. Yellow phosphorus vapor when wet air can be oxidized to phosphoric acid and phosphoric acid.
national health standard provisions: Workplace air yellow phosphorus, the maximum allowable concentration 0.03mg/m3.
chronic phosphorus poisoning caused by inhalation. An early manifestation of rhinitis, laryngitis, bronchitis, and accompanied by a neurasthenic syndrome. Progress of the disease, there toothache, tooth loosening, gingivitis, alveolar abscess, x-ray jaw piece showed, osteoporosis, decalcified, and necrosis. And may have liver damage and anemia.
Thephosphorus in poisoning (phosphorus poisoning)
oral, respiratory tract or skin absorb a certain amount of phosphorus, cause digestive, respiratory and bone lesions of the phenomenon. In severe cases of acute poisoning can cause acute hepatic necrosis. Chronic poisoning can occur in severe osteoporosis and necrosis, in particular the following jaw more common. Skin contact can cause burns.
phosphorus through the respiratory tract, gastrointestinal tract or skin into the body, most elements in the form of existence, a small part of the oxidation of phosphorus hypoxia compounds circulating in the blood and deposition in the liver and bone. A small amount of discharge from the breath, sweat and faeces. Yellow phosphorus, phosphoric acid amount in the body increase, accelerate the excretion of calcium, causing bone decalcification; can inhibit the oxidative process caused by protein and fat metabolism disorders, liver glycogen to reduce lower blood sugar, blood lactic acid increased. The pathological anatomy of visible central nervous system is a bit flaky bleeding, pericardial and endocardial haemorrhagic, gastrointestinal bleeding, cardiac and liver fatty change or necrosis.
acute yellow phosphorus poisoning is diagnosed based on exposure history and clinical manifestations. The diagnosis of chronic yellow phosphorus poisoning, in addition to the history of exposure and clinical manifestations, still should be done mandible X-ray film.
yellow phosphorus is phosphate ore manufacturing phosphate intermediate products or by-products. For manufacturing red phosphorus oxide phosphorus, phosphoric acid, fireworks, firecrackers, flares and other industrial and military.
phosphorus yellow phosphorus, red phosphorus, violet phosphorus and black phosphorus four isomers. Yellow phosphorus (white phosphorus) is highly toxic and can be deadly when the absorption capacity of 1mg/kg body weight. Yellow phosphorus concentration of 0.2 ~ 1.2mg/m3 production environment, workers, chronic yellow phosphorus poisoning. Red phosphorus (also known as red phosphorus) less toxic. Purple phosphorus and black phosphorus is very rare.
treatment of oral poisoning, immediately with 0.2% copper sulphate gastric lavage, or 0.1% potassium permanganate gastric lavage. Repeatedly washed until the wash liquid phosphorus free until the garlic flavor. After gastric lavage, oral administration of 0.5% to 1.0% copper sulfate 4mL every 15min for a total of 2 to 3 times. Oral administration of 33% sodium sulfate 30mL catharsis. Fasting milk and fat. Poisoning by inhalation, intravenous injection of l% glucose and vitamin C to protect the liver and heart. Shock and circulatory failure appears to be symptomatic. Yellow phosphorus skin burns, immediately flooding. Before the formation of phosphoric acid and 2% sodium bicarbonate rinse, and then 1% copper sulfate rinse, residual particles of yellow phosphorus to the formation of phosphide of copper. Patients with chronic poisoning, removed from the original job, give symptomatic treatment.
acute inhalation toxicity in patients with mild headache world pie network information , dizziness, general weakness, vomiting, and sometimes bradycardia. Sustainable few days later, gradually being restored. Patients with moderate poisoning, the symptoms are serious, heart rate, low blood pressure. 2 to 3 days later, jaundice and hepatomegaly. Severe poisoning can cause acute liver necrosis and coma. Also reported in the literature, yellow phosphorus liquid skin burns caused by acute hemolytic anemia, leading to acute renal failure.
oral poisoning can occur, nausea, vomiting, mouth and stomach burning sensation, as well as abdominal pain, diarrhea, and so on. A few days, jaundice and hepatomegaly may develop acute liver necrosis World pipe network reported that the world's steel pipe network to provide the world's steel pipe network editor . The most severe cases, within hours, by the excitement transferred suppression, coma, circulatory failure, and death.